Abstract

There are short-term and long-term variations seen in blood pressure. An abnormal pattern in cyclic variations of blood pressure (diurnal or seasonal) correlates well with an increased cardiovascular risk for hypertension, regardless of the resting blood pressure level. Increased blood pressure reactivity to smoking is also associated with a higher cardiovascular risk, and may be a significant determinant of cyclic blood pressure variations. The purpose of the study was to compare systolic (SBP) and diastolic blood pressure (DBP) between smokers and non smokers and elicit the cardiovascular risk. To achieve the purpose twenty (20) male athletes were selected from Dept. of physical education and sports science, Annamalai University, among these selected subjects ten (10) were smokers and ten (10) were non smokers. The parameters analyzed were 24-hour daytime and nighttime systolic and diastolic blood pressure using sphygmomanometer. The data was collected at six different times of the day 02:00, 06:00, 10:00, 14:00, 18:00 and 22:00 hours respectively. The data was collected from smokers and non smokers on SBP and DBP and 2 × 6 ANOVA with last factor repeated was computed. SBP and DBP showed significant variations between smokers and non smokers. SBP and DBP showed significant variations between smokers and non smokers at different times of the day. Not only the absolute value of blood pressure, but also the patterns of its excessive variation are closely related to the occurrence of cardiovascular events, so these patterns are clinically important. It may be difficult to explain the exact mechanisms of various abnormal short-term and long-term blood pressure variations at present. Smoking has been suggested as the causes of abnormal variations in blood pressure.

Keywords

Systolic Blood Pressure, Diastolic Blood Pressure, Rhythm, Handball, Players,

References

  1. D. A. Sica, What are the influences of salt, potassium, the sympathetic nervous system, and the renin angiotensin system on the circadian variation in blood pressure? Blood Pressure Monitoring, 4 (1999) 9-16.
  2. K. Narkiewicz, M. Winnicki, K. Schroeder, B. G. Phillips, M. Kato, E. Cwalina, V. K. Somers, Relationship between muscle sympathetic nerve activity and diurnal blood pressure profile, Journal of Hypertension, 39 (2002) 168-72.
  3. F. Portaluppi, B. Bagni, E. degli Uberti, L. Montanari, R. Cavallini, G. Trasforini, A. Margutti, M. Ferlini, M. Zanella, M. Parti, Circadian rhythms of atrial natriuretic peptide, rennin, aldosterone, cortisol, blood pressure and heart rate in normal and hypertensive subjects, Journal of Hypertension, 8 (1990) 85-95.
  4. S. J. Kim, H. S. Kang, S. J. Hwang, I. S. Sohn, C. W. Choue, K. S. Kim, J. S. Song, J. H. Bae, The relation of circadian blood pressure variation to left ventricular mass, diastolic function, and dynamic QT dispersion, Korean Circulation Journal, 35 (2005) 382-388.
  5. K. Kario, K. Shimada, Risers and extreme-dippers of nocturnal blood pressure in hypertension: antihypertensive strategy for nocturnal blood pressure, Clinical and Experimental Hypertension, 26 (2004) 177-189.
  6. K. Kario, T. G. Pickering, Y. Umeda, S. Hoshide, Y. Hoshide, M. Morinari, M. Murata, T. Kuroda, J. E. Schwartz, K. Shimada, Morning surge in blood pressure as a predictor of silent and clinical cerebrovascular disease in elderly hypertensives: a prospective study, Circulation, 107 (2003) 1401- 1406.